IGF-1 LR3 Peptide

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Iyo Insulin-yakafanana neKukura Factor (IGF-1 LR3 Peptide) ndeimwe yemahomoni e endocrine anogadzirwa muchiropa. Kuburitswa kweiyi hormone kunowedzera pamberi peHuman Growth Hormone.


Product Sumo:

Iyo Insulin-yakafanana neKukura Factor (IGF-1 LR3 Peptide) ndeimwe yemahomoni e endocrine anogadzirwa muchiropa. Kuburitswa kweiyi hormone kunowedzera pamberi peHuman Growth Hormone. Masero mazhinji mukati memhasuru dzemuviri wemunhu akashongedzerwa nema cell receptors ane hukama hwepamusoro hweInsulin-sekukura chinhu. Izvi zvinoita kuti iyi hormone ive imwe yeakanakisa kukura mahomoni uye kufambisa kwesero kukura kwaanoita nekutarisa akasiyana chaiwo matissue uye mune mamwe autocrine cell kutaurirana maitiro, inofambisa kupatsanurwa kwesero.


ZVINOBATSIRA IGF-1 LR3 Peptide:


Inofambisa protein synthesis mumuviri.


Inogadzirisa kuchengetwa kwemafuta uye nzira kuti ishandiswe kugadzirwa kwesimba. Izvi zvinokonzera kurasikirwa kwemafuta kunooneka.


Inokurudzira migumisiro yakanaka pametabolism; kuwedzera kuonda kwemuviri uye kuderedza mafuta


Zvinowedzera regenerative ehupfumi yemuviri tsinga tishu.


Inosimudzira anti-oxidant kubatsirwa uye ligament simba.


Inowedzera hyperplasia mumasero emhasuru, iyo inotungamira kune yakazara tsandanyama tishu.


Iyo hafu yehupenyu yeIGF-1 LR3 inenge 20 - 30 maawa. Iyo ine simba kupfuura yakajairika base IGF-1.


Nekuti inogona kuchengetedzwa mumuviri kweanopfuura zuva, inosunga nema cell receptors mumasero emhasuru uye inomutsa kutaurirana kwesero izvo zvinozovandudza kukura kwemhasuru zuva rese.


IGF-1 LR3 inhibits the movement of glucose into the body cells which facilitates fat burning and the use of fat in the body for the production of energy. Its long life of close to a day has made it a preferred variant by a majority of patients and physicians because site injections are never necessary. IGF-1 LR3 cycles the whole body and binds to the receptors on muscle cells then acts for about a day, so a daily administration of this dosage is strongly supported.


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